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. 2020 Jan 22;82(2):17. doi: 10.1007/s11538-019-00685-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 13 — For the symplast–apoplast model, the mechanisms of auxin transport and PIN localisation determine the steady-state pattern in auxin distribution. a For model (1),(6)–(8)b),d) considering non-saturating auxin flux and flux-induced PIN localisation passage and spot patterns emerge similar to model (1)–(3). b For model (1),(6)–(8)a),d) considering saturating auxin flux and flux-induced PIN localisation similar patterns to the case with non-saturating auxin flux and flux-induced PIN localisation. c For model (1),(6)–(8)a),c) considering saturating auxin flux and concentration-induced PIN localisation a pattern of single-cell spots with high auxin concentration emerges. For model (1),(6)–(8)b),c) considering non-saturating auxin flux and concentration-induced PIN localisation the steady-state distribution is homogeneous (not shown). All parameters are described in Appendix Tables 2 and 4, with periodic boundary conditions (Color figure online)