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. 2019 Oct 23;71(3):970–985. doi: 10.1093/jxb/erz468

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© The Author(s) 2019. Published by Oxford University Press on behalf of the Society for Experimental Biology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Fig. 2. — CCA1 is a repressor of BAS1 and SOB7 expression. Ectopic expression of pBAS1:BAS1-GUS (A) and pSOB7:SOB7-GUS (B) in the cca1-1 background caused BR deficiency-associated dwarfism in about a quarter of T₁ plants of both transgenic events. Single-locus T-DNA insertional pBAS1:BAS1-GUS/cca1-1 and pSOB7:SOB7-GUS/cca1-1 homozygous T₃ lines were selected from BR-dwarf plants and crossed with Col-0, respectively. Homozygous pBAS1:BAS1-GUS (C) and pSOB7:SOB7-GUS (D) sibling lines in cca1-1 and Col-0 backgrounds were selected from the two F₂ segregation populations for comparison of morphology. F₃ homozygous segregants of pBAS1:BAS1-GUS/Col-0 (E), pBAS1:BAS1-GUS/cca1-1 (E), pSOB7:SOB7-GUS/Col-0 (F), and pSOB7:SOB7-GUS/cca1-1 (F) retained their morphologically normal or BR-dwarf phenotypes, respectively.