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. 2020 Jan 17;9:1526. doi: 10.3389/fonc.2019.01526

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Copyright © 2020 Ros, Pegoraro, De Angelis, Sgarra, Zucchelli, Gustincich and Manfioletti.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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HMGA2-AS1 lncRNAs regulate cell migration ability through HMGA2. Transwell assay in PANC1 cells transfected with empty vector (pEGFP-N1) or with a vector expressing HMGA2 (pEGFP-N1-HMGA2), treated with siCTRL or siHMGA2-AS1-all. The data are presented in pEGFP-N1 and in pEGFP-N1-HMGA2 conditions as the mean of the percentage of siHMGA2-AS1-all migrated cells relative to the siCTRL ± SD (n = 3) set to 100% in each condition. Lower part, a representative western blot of HMGA2 overexpression and endogenous protein is presented. β-actin was used as a loading control. **p ≤ 0.01, NS: Not Significant; two-tailed Student's t-test. Also see uncropped figure scan in Supplemental Images 7–9.