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. 2020 Jan 21;9:1565. doi: 10.3389/fonc.2019.01565

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Copyright © 2020 Wang, Deng, Dai, Niu and Zhou.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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miR-27a inhibited tumor growth of cSCC in vivo. (A,B) Growth curves of tumor volumes in miR-27a mimic group and NC mimic group were determined every 3 days. Representative photographs of tumors were shown below. (C,D) Relative expression of miR-27a in HSC-5 cells treated with miR-27a mimic or negative control. (E,F) miR-27a overexpression regulated the phosphorylation of EGFR and the phosphorylation of p65, IκB, and IKK. Each experiment was performed in triplicate and data are presented as mean ± s.d. One-Way ANOVA and Dunnett's Multiple comparison test were used to analyze the data (*P < 0.05, **P<0.01, ***P <0.001).