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. 2020 Jan 30;11:619. doi: 10.1038/s41467-020-14373-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a LCN2 in non-primarily neurodegenerative and non-ischemic neuropsychiatric diseases (ND, n = 73), Alzheimer’s disease (AD, n = 47), vascular dementia (VaD, n = 27), mixed Alzheimer’s and vascular dementia (MD, n = 31), Lewy body dementias (LBD, n = 36), frontotemporal dementia (FTD, n = 21), and sporadic Creutzfeldt-Jakob disease (CJD, n = 54). Results are shown as mean ± SD for each condition. b Area under the curve (AUC) derived from receiver operating characteristic (ROC) curves, Standard Error (SE), 95% Confidence interval (95% CI) for LCN2 in the comparative analyses. c ROC curves of differentiating ND and VaD as well as AD and VaD. Differences between groups were analysed with Tukey contrasts using linear regression models controlled for age and sex. *p < 0.05, **p < 0.01, ***p < 0.001.