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. 2020 Jan 30;15:6. doi: 10.1186/s13027-020-0272-3

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© The Author(s). 2020

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — The activation of NOD1 increased CHX-induced apoptosis. NOD1 expression increased in SiHa cells treated with iE-DAP (**P < 0.005, ***P < 0.0001) (a). The NOD1 activation enhanced CHX-induced apoptosis. HPV16-positive SiHa cells were treated with iE-DAP in the presence or absence of CHX. Cell viability was measured by flow cytometry. The apoptosis of SiHa cells increased significantly in the presence of CHX (P < 0.005) (b)