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. 2020 Jan 14;11(6):1457–1467. doi: 10.7150/jca.35479

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This is an open access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/). See http://ivyspring.com/terms for full terms and conditions.

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IL1A mediated the oncogenic activity of p16 in cervical carcinoma cell lines: (A) Ca Ski Cells were transfected with independent siRNA against Non-Target control or IL1A. IL1A transcript levels were determined by Taqman qRT-PCR analyses. IL1A knockdown affected cell viability (B) and proliferation (C)(D) of Ca Ski. (E) IL1A transcript levels of Ca Ski ctrl-sh-ctrl, Ca Ski ctrl-sh-p16#1, Ca Ski ctrl-sh-p16#2, Ca Ski IL1A-sh-p16#1 and Ca Ski IL1A-sh-p16#2 were determined by Taqman qRT-PCR analysis. IL1A overexpressing affected the cell viability (F) and proliferation (D)(H) of p16 silencing Ca Ski.