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. 2020 Feb 3;8:4. doi: 10.1038/s41413-019-0081-8

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Activation of Wnt7b in aged mice increases bone mass. a RT-qPCR analyses of Wnt7b mRNA in the bones of mice treated with Dox for 2 weeks starting at 4 weeks of age. Ctrl: Osx-rtTA;R26-Wnt7b mice; Wnt7b: Osx-rtTA;tetO-Cre;R26-Wnt7b mice. *P < 0.05, n = 3. b Whole-body (excluding head) DXA analyses of bone mineral content (BMC) and bone mineral density (BMD) of Osx-rtTA;tetO-Cre;R26-Wnt7b mice with or without Dox treatment from 15 through 16 months of age. *P < 0.05, one-way ANOVA, n = 5 (four males, one female). c Contact X-ray images representative of the hindlimbs at time of sacrifice. Error bars: stdev in all figures.