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. 2020 Feb 3;25:4. doi: 10.1186/s11658-020-0201-x

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© The Author(s) 2020

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — MiR-515-5p is a target of LINC00511 in gastric cancer. a The recognized binding sites between LINC00511 and miR-515-5p. b MiR-515-5p expression in 25 pairs of gastric cancer tissues (GC tissues) was evaluated using quantitative RT-PCR. c Low miR-515-5p levels correlated with poor survival in gastric cancer patients based on the TCGA dataset. d MiR-515-5p overexpression led to a reduction in luciferase activity in the LINC00511-Wt group. e LINC00511 knockdown increased miR-515-5p expression in BGC823 cells. f The RIP assay revealed that both LINC00511 and miR-515-5p expression are enhanced in the mixture immunoprecipitated by anti-Ago2. g LINC00511 expression negatively correlated with miR-515-5p expression in gastric cancer tissues. *p < 0.05