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. 2018 Nov 5;9(1):1699–1717. doi: 10.1080/21505594.2018.1532243

Figure 2.

Figure 2.

Binding of H. pylori pediatric strains to gastric mucins according to their babA and sabA genotype. A-C) Binding of NUD and PUD H. pylori babA+ and babA− strains (NUD babA−, n = 7; PUD babA−, n = 3; NUD babA+, n = 2; PUD babA+, n = 8) to mucins isolated from human tumor (A), human normal (B) and monkey normal (C) gastric tissue. D-F) Binding of NUD and PUD H. pylori sabA+ and sabA− strains (NUD sabA off, n = 3; PUD sabA off, n = 6; NUD sabA on, n = 6; PUD sabA on, n = 5) to mucins isolated from human tumor (D), human normal (E) and monkey normal (F) gastric tissue. Results are expressed as minimum, first quartile, median, third quartile, and maximum in a box and whisker plot, after subtraction of the background signal (no mucin) at each pH. Mann-Whitney U test comparing binding to mucins of NUD vs PUD strains with the same genotype, * p ≤ 0.05, ** p ≤ 0.01.