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. 2020 Jan 16;21(3):1509–1516. doi: 10.3892/mmr.2020.10944

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Copyright: © Lang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — miR-149 is involved in BCYRN1-regulated glycolysis. (A) Predicted binding site between BCYRN1 and miR-149. (B) miR-149 expression levels following transfection with miR-149 mimic or miR-149 inhibitor. (C) Luciferase assay suggested a relationship between BCYRN1 and miR-149 expression. (D) miR-149 expression levels were regulated by BCYRN1. miR-149 knockdown rescued the si-BCYRN1-induced decrease in (E) glucose consumption and (F) lactate production compared with the si-BCYRN1 group. *P<0.05 vs. si-NC or NC; ^#P<0.05 vs. si-BCYRN1. BCYRN1, brain cytoplasmic RNA 1; miR, microRNA; NC, negative control; si, small interfering RNA.