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. 2020 Feb 8;19:13. doi: 10.1186/s12933-020-0994-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Echocardiographic parameters: Representative parasternal short axis views of the LV (Bmode) in diastole (a) and systole (b) 1-week post LAD ligation. The anterolateral infarct is outlined with dotted yellow lines. M-mode imaging (c) reveals thinning of the left ventricular anterior wall (LVAWd), increased internal diameter (LVIDd) and reduced function. d–f represent quantitation of LV internal diameter in diastole, LV internal diameter in systole and fractional shortening. MI reduced FS and led to ventricular dilatation. LVIDd was reduced by empagliflozin therapy. *p < 0.05 MI versus sham, ^†p < 0.05 versus MI control