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(A) Fresh-PRP and (B) FD-PRP induce the phosphorylation and activation of ERK in a concentration-dependent manner, which is inhibited in the PDGFR knockdown line. Results are derived from three independent experiments performed in triplicate (n=3). PRP, platelet-rich plasma; FD-PRP, freeze-dried platelet-rich plasma; ERK, extracellular signal-regulated kinase; PDGFR, platelet-derived growth factor receptor.
