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. 2020 Feb 10;10:2257. doi: 10.1038/s41598-020-59039-7

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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(a) Plasma corticosterone levels with and without the FST 30 min after i.p. administration of YL (3–30 mg/kg, n = 11–12). (b) Hypothalamic CRH mRNA expression normalized to β-actin with and without the FST after i.p. administration of YL (30 mg/kg, n = 4). Values are expressed as the mean ± S.E.M. *P < 0.05 compared with each group by ANOVA followed by Tukey-Kramer’s (a) or nonparametric Mann–Whitney U (b) tests.