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. 2020 Jan 27;141(6):418–428. doi: 10.1161/CIRCULATIONAHA.119.043132

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© 2020 The Authors.

Circulation is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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Figure 3. — Composition of LQTS-specific genetic panels. Percentage of genetic panels including genes previously reported as causing LQTS. *Genes with definitive evidence for causality of typical LQTS. †Genes with strong or definitive evidence for causality in LQTS with atypical features. ‡Genes with definitive evidence for causality in multiorgan including QT prolongation but only moderate or limited evidence for isolated LQTS. §Genes with strong level of evidence for causality in acquired LQTS but only limited or disputed evidence for congenital LQTS.