Table 1.
Definitions [25].
| Term | Definition |
|---|---|
| Passive transmural drainage | The insertion of transmural endosthesis without the nasocystic drain in order to enable an outflow of necrotic content through the fistula and keeping the fistula unobstructed. |
| Active transmural drainage | The insertion of endoprostheses along with nasocystic drain through transmural fistula to the lumen of collection in order to enable WOPN flush. |
| Single transluminal gateway technique (SGT) | Complete removal of necrotic tissues through a single fistula created between the cavity of necrotic collection and the lumen of gastrointestinal tract (stomach or duodenum). Endoprosthesis and drains were inserted through the single fistula in the case of unilocular necrotic collections. |
| Multiple transluminal gateway technique (MTGT) | The creation of multiple transmural tracts between the gastrointestinal lumen and the WON cavity. In MTGT another transmural tract between the necrotic cavity and the gastrointestinal lumen was performed in case of multilocular necrotic collections divided by septa. |
| Single transluminal gateway transcystic multiple drainage (SGTMD) | Additional transmural drainage of extensive necrosis through a single fistula. Stents and nasocystic drains were introduced in the subcavities of WOPN through the single transmural tract and canals between necrotic subcavities. |
| Passive transpapillary drainage | The insertion endoscopic pancreatic stent to pancreatic duct through duodenal papilla. |
| Active transpapillary drainage | Endoscopic insertion pancreatic endoprosthesis as well as nasal drain to pancreatic duct through duodenal papilla. |
| The therapeutic success of WOPN | The absence of symptoms and complete regression of the necrotic collection. |
| The success of endotherapy of MPD disruption | The lack of contrast flow outside the MPD in patients with WOPN treated endoscopically for MPD disruption |
| Long-term success of WOPN treatment | The therapeutic success of WON endotherapy, the success of endotherapy of MPD disruption, and the lack of recurrence of PFCs during follow-up. |