Figure 1.

PR-AKI can have prerenal, intrinsic, and post-renal etiologies. Prerenal causes may be secondary to hemorrhage, hypovolemia (¹from hyperemesis gravidarum), sepsis, or congestive heart failure. Intrinsic renal causes include acute tubular necrosis, renal cortical necrosis, thrombotic microangiopathy, preeclampsia spectrum disorders, acute fatty liver of pregnancy, glomerulonephritis, or acute interstitial nephritis (²from medication exposure). Postrenal etiologies include mechanical obstruction (³postsurgical), neoplasm, or uteropelvic (⁴obstruction from pregnancy).