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. 2020 Feb 7;11:33. doi: 10.3389/fphys.2020.00033

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Copyright © 2020 Wu, Lin, Lin, Rao, Lin, Wu and Li.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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(A) Representative recordings of LV pressure-time curves of each group after 4-week pharmacological treatment. (B) Hemodynamic properties obtained from each group. Lower SBP accompanied by lower RPP was found in the ICM group than in the Sham group, while ischemic injury did not change DBP statistically. Dex restored the SBP in the ICM + Dex group. Persistent ischemia damaged cardiac contraction, manifesting as reduced Max Pressure, Max dP/dt, and Min dP/dt and increased EDP. Dex increased Max Pressure, Max dP/dt, and Min dP/dt and reduced EDP in the ICM + Dex group when compared to the ICM group. BML weakened the effects of Dex on left ventricular contraction. Data are expressed as mean ± SD. *p < 0.05 vs. Sham group, ^#p < 0.05 vs. ICM group, ^▲p < 0.05 vs. ICM + Dex group (n = 8 for each group).