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. 2020 Feb 18;11(2):125. doi: 10.1038/s41419-020-2324-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — a Various dosages of GSK2656157 was pretreated cells for 1 h, then stimulation with 100 ng/ml HMGB1 for 48 h. ATF4 and SESN2 in DC2.4 cells were determined by Western blotting. b, c After induction with siRNA or LV-RNA for 72 h, DC2.4 cells were treated with 100 ng/ml HMGB1 for 48 h, then SESN2 as well as ERS apoptotic-related protein CHOP were examined by Western blotting. β-actin served as the internal standard. Data were presented as the mean ± SD, n = 3 per group. Statistical significance: *P < 0.05 versus the scramble group; ^#P < 0.05 versus the scramble group treated with HMGB1; ^&P < 0.05 versus the scramble group treated with HMGB1 and GSK2656157.