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. 2020 Feb 18;11(2):131. doi: 10.1038/s41419-020-2326-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a Western blotting was applied to analyze inflammation signaling pathways-related proteins expressions in AAA tissue at indicated time points. JAK2, STAT3, p-STAT3, NF-κB, and p-NF-κB expressions were evaluated in AngII⁺ group and compared with AngII⁺ BP-1-102⁺ group (AngII⁺BP⁺). AngII⁺ BP-1-102⁺ group showed reduced inflammation signaling pathways-related proteins compared with AngII⁺ group at each time point. Saline treated group was used as control (mock). b Relative protein levels of JAK2, STAT3, p-STAT3, NF-κB, p-NF-κB were determined by normalized with β-actin expression in AngII⁺ group and AngII⁺ BP-1-102⁺ group (AngII⁺BP⁺) at indicated time points. Saline treated group was used as control (green column). AngII⁺ BP-1-102⁺ group (blue column) showed reduced inflammation signaling pathways-related proteins compared with AngII⁺ group (red column) at each time point. The results were presented as mean ± standard deviation (SD). n = 8. *P < 0.01 vs. AngII⁺ group (two-way ANOVA followed by Tukey’s test). NS indicates non-significant differences compared with AngII⁺ group.