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. 2019 May 30;69(3):502–512. doi: 10.1136/gutjnl-2019-318320

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© Author(s) (or their employer(s)) 2020. Re-use permitted under CC BY. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution 4.0 Unported (CC BY 4.0) license, which permits others to copy, redistribute, remix, transform and build upon this work for any purpose, provided the original work is properly cited, a link to the licence is given, and indication of whether changes were made. See: https://creativecommons.org/licenses/by/4.0/.

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(A and B) Correlation graphs of changes between weeks 0 and 2 in relevant clinical variables and faecal microbiota operational taxonomic unit (OTUs) for both groups. A shows RYGB-R and B shows METS-R. Only significant (Spearman) correlations with p<0.05 are shown. These correlations have not been corrected for false discovery. Colours indicate increase (blue) or decrease (red). CCL2, chemokine ligand 2; CD68, cluster of differentiation 68; EGP suppression, suppression of endogenous glucose production by insulin (hepatic insulin sensitivity); HDL, high-density lipoprotein; IL 6, interleukin 6; LDL, low-density lipoprotein; LPS, lipopolysaccharide; METS-R, metabolic syndrome recipient; Rd, rate of glucose disappearance (peripheral insulin sensitivity); RYGB-R, Roux-en-Y gastric bypass recipient; TNF alpha, tumour necrosis factor alpha.