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. 2020 Feb 21;10:3153. doi: 10.1038/s41598-020-59917-0

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The inflammatory response to Aβ_1–42 is attenuated in IFNAR1^−/− primary microglia. Transcript levels of (a) IL1β, (b) IL6 and (c) TNFα were analysed by QPCR analysis following 10 μM monomeric Aβ_1–42 treatment (24–72 hours). Collected media was also analysed via ELISA for the corresponding cytokines (d) to (f). Data is displayed as mean ± SEM (*p < 0.05, 2-way ANOVA, Sidak’s multiple comparisons test, wild type vs. IFNAR1^−/–, n = 5).