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. 2020 Feb 21;11:995. doi: 10.1038/s41467-019-14275-y

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© The Author(s) 2020, corrected publication 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — The CD score was calculated using 147 CD risk alleles and the UC score using 119 UC risk alleles. Both scores were generated for a discovery cohort comprising 99 VEO-IBD cases (VEO-COLORS), 7578 CD cases, 6318 UC cases, 18,780 UK population controls (from the UKIBDGC), all of European ancestry. The replication cohort comprised 117 VEO-IBD cases (VEO Toronto) and 2603 population controls (from the NIDDK Genetics Consortium). The CD and UC risk scores did not significantly differ between the two VEO-IBD cohorts (CD P = 0.98; UC P = 0.64). The Student’s t-test was used in group comparisons.