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. 2020 Feb 1;17(3):910. doi: 10.3390/ijerph17030910

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© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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PTP4A3 participated in benzene toxicity under the regulation of HIF-1alpha. (a) The mRNA level of PTP4A3 was measured with real-time quantitative PCR. (b,c) The expression of PTP4A3 in benzene-exposed mice was measured with western blot. (d) The relationship between HIF-1alpha and PTP4A3 in benzene exposed mouse bone marrow was measured with ChIP-qPCR. (e,f) The expression of PTP4A3 was measured in HIF-1alpha high expression K562 cells (K562-HIF-1α⁺). The β-actin was applied to internal reference. *: p < 0.05, compared with the control group.