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. 2020 Jan 23;19(3):2422–2430. doi: 10.3892/ol.2020.11338

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Copyright: © Sumransub et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Tumor cell apoptosis caused by effector T cells activated by BCA55-121-WP-RNA-pulsed DCs and those activated by BCA55-121-CSC-RNA-pulsed DCs. Cancer cell apoptosis was assessed by an immune cell killing assay using the IncuCyte^® live cell analysis system. Time-dependent cancer cell apoptotic count induced by total lymphocytes against (A and B) BCA55-121-WP and BCA55-121-CSC compared with the apoptotic cell count of each condition at 0 h. The y-axis represents the numbers of apoptotic cells compared with those in the control group at the starting point of each treatment condition. The data are from triplicate wells of one experiment. *P<0.05 vs. T cells from the unpulsed DCs. CSC, cancer stem cell; DCs, dendritic cells; WP, whole population.