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. 2020 Jan 23;19(3):2265–2271. doi: 10.3892/ol.2020.11334

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — miR-202 inhibits cell proliferation and cell glycolysis in HCC. miR-202 expression was detected using reverse transcription-quantitative PCR following (A) 97-L and (B) Huh7 cell transfection with miR-NC, miR-202 mimic or miR-202 inhibitor. Cell Counting Kit 8 cell proliferation assay was used to evaluate cell proliferation following (C) 97-L and (D) Huh7 cell transfection with miR-NC, miR-202 mimic or miR-202 inhibitor. (E) Relative glucose consumption was revealed following 97-L and Huh7 cell transfection with miR-NC, miR-202 mimic or miR-202 inhibitor. (F) Relative lactate production was revealed following 97-L and Huh7 cell transfection with miR-NC, miR-202 mimic or miR-202 inhibitor. Data are presented as the mean ± SD from three independent experiments. *P<0.05 vs. corresponding control. miR, microRNA; HCC, hepatocellular carcinoma; NC, negative control; OD, optical density; inh, inhibitor.