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. 2020 Jan 17;19(3):2272–2280. doi: 10.3892/ol.2020.11313

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Copyright: © Liang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Knockdown of MARCKSL1 suppresses epithelial-mesenchymal transition-associated proteins and AKT activation. Expression of E-cadherin, N-cadherin, vimentin, SNAI2, p-AKT and AKT was evaluated by western blotting. GAPDH served as loading control. Multiple bands were observed for E-cadherin and N-cadherin due to post-translational modifications as previously described (67,68). MARCKSL1, MARCKS like 1; AKT, protein kinase B; si, small interfering; SNAI2, snail family transcriptional repressor 2; p, phosphorylated.