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. 2020 Feb 26;15(2):e0229671. doi: 10.1371/journal.pone.0229671

Fig 2. Dop1R1 receptor expressed in MB modulates larval locomotion.

Fig 2

A. Pan-neuronal expression of an RNAi against Dop1R1 (elav>RNAiDop1R1) results in increased motor behavior as compared to genetic controls. B. Expression of RNAiDop1R1 in the entire MB neuronal population (OK107>RNAiDop1R1) or C. in the γ-lobe forming neurons (201y>RNAiDop1R1), induces an increase in larval locomotion. D. No effect on locomotion is observed when the RNAi is expressed in the α′β′ MB forming neurons (c305a>RNAiDop1R1). In each case, genetic controls are animals bearing one copy of the Gal4 driver or the undriven UAS-RNAi Dop1R1 (in white and gray bars respectively). Data represent results obtained from at least 34 different larvae per experimental condition. * and *** indicates p<0.05 and p<0.005 compared to genetic controls; one-way ANOVA followed by Tukey post-hoc test.