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. 2020 Feb 27;10:22. doi: 10.1186/s13578-020-00386-3

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Suggested role of SOD3 and MSCs in various conditions. SOD3 found to increase survival of MSCs against starvation-induced limited survival. Autophagy in MSCs found to be regulated in relation to SOD3 and overexpression of SOD3 exhibited increase autophagy. MSCs-secreted SOD3 found to regulate differentiation and maintain phenotype features. SOD3 in MSCs exhibited increase protection against oxidative damage and improves the therapeutic potential of MSCs