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. 2020 Feb 27;11(2):153. doi: 10.1038/s41419-020-2353-z

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Cells were treated with OTA or/and CsA for 48 h. A total of 5 mM 3-MA as an inhibitor of autophagy was used at the same time. Relative mRNA levels of genes a were tested. Nephrotoxicity related proteins and autophagy-related proteins b–i were measured. Immunofluorescence staining of vimentin and α-SMA j was performed by laser confocal microscopy. All results were expressed as means ± SD (n = 3). Significance compared with the control group, *P < 0.05; significance of combination group compared with the combination group with 3-MA, ^#P < 0.05.