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. Author manuscript; available in PMC: 2021 Jun 1.
Published in final edited form as: Transl Stroke Res. 2019 Aug 31;11(3):418–432. doi: 10.1007/s12975-019-00729-4

Fig. 2. Activation of PKCε increases serine phosphorylation of the MAS components GOT2 and OGC in synaptosomes.

Fig. 2

Synaptosomes from rat cortices were isolated 48 hours after TAT or ΨεRACK (0.2 mg/kg i.p.) treatment. Proteins were immunoprecipitated with (a) phospho-serine, (b) phospho-threonine, (c) phospho-tyrosine, and immunoblotted for GOT2 and OGC. Input-GOT2 acts as a loading control. Quantification fold change of TAT control and representative blot as shown above (n=4–5, mean ± SEM, * P <0.05, ** P <0.01, ns = non-significant, Two-tailed unpaired Student’s t-test). GOT2: Glutamic oxaloacetic transaminase 2; OGC: Oxoglutarate carrier.