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. 2020 Feb 28;10:3767. doi: 10.1038/s41598-020-60775-z

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The genome-wide profiles of H3K4me3 and H3K27ac ChIP-seq peaks in neuronal cells, non-neuronal cells and bulk brain. (a) Genomic annotation of H3K4me3 and H3K27ac profiles in neuronal and non-neuronal cells. Each ChIP-seq peak was analyzed by CEAS. (b) The top 10 significantly enriched GO terms (y-axis) and their p-values (x-axis) of differentially modified genes. Only the terms with statistical significance (P < 0.01) are shown, such that no terms are shown for H3K4me3 ChIP-seq of non-neuronal samples. (c) Clustering analysis of ChIP-seq peak in neuronal, non-neuronal cells and bulk brain. (d) Principal component analysis of ChIP-seq peak in neuronal, non-neuronal cells and bulk brain.