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. 2020 Feb 28;11(2):156. doi: 10.1038/s41419-020-2351-1

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a Western blot of endogenous coIP of MCL1 (IP) with P18 (IB), BAX (IB), and P18 (IB) in WAC2. b Western blot of in vitro pulldown of recombinant proteins (IP), ANK1–4, ANK4–5, P18, p16, and chimera, with recombinant MCL1 (rMCL1) (IB). Cartoon of the p16, P18, and chimera proteins are shown below the blot. rBH3-containing helix is highlighted in yellow (Residues: 150–161). c Western blot of in vitro pulldown of recombinant P18 (IP) with rMCL1 (IB) with and without MCL1 inhibitor S63845.