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. 2020 Jan 21;11(3):526–536. doi: 10.1111/1759-7714.13247

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© 2019 The Authors. Thoracic Cancer published by China Lung Oncology Group and John Wiley & Sons Australia, Ltd

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.

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ACTL8 regulates angiogenesis in human LUAD cells. (a) Infection with lenti‐shACTL8 for 24 hours abolished the A549 cell‐induced increase in tube formation. (b) Infection with lenti‐shACTL8 for 24 hours had no effect on the NCI‐H1975‐induced increase in tube formation. The area of the tube branches was calculated and quantified. The data are presented as the mean ± standard deviation; n = 3. *P < 0.05 and ***P < 0.001 versus lenti‐shCtrl. ACTL8, Actin‐like protein 8; LUAD, lung adenocarcinoma; sh, short‐hairpin; lenti, lentivirus; Ctrl, control. () shCtrl and () shACTL8.

Inline graphic — ACTL8 regulates angiogenesis in human LUAD cells. (a) Infection with lenti‐shACTL8 for 24 hours abolished the A549 cell‐induced increase in tube formation. (b) Infection with lenti‐shACTL8 for 24 hours had no effect on the NCI‐H1975‐induced increase in tube formation. The area of the tube branches was calculated and quantified. The data are presented as the mean ± standard deviation; n = 3. *P < 0.05 and ***P < 0.001 versus lenti‐shCtrl. ACTL8, Actin‐like protein 8; LUAD, lung adenocarcinoma; sh, short‐hairpin; lenti, lentivirus; Ctrl, control. () shCtrl and () shACTL8.