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. 2020 Feb;30(2):263–275. doi: 10.1101/gr.253328.119

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© 2020 Gong et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genome.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 6. — Analyses of key signaling pathways related to liver development. (A) Changes in effectors of key signaling pathways based on KEGG and other studies (Yimlamai et al. 2014) (TFs, proteins, or RNAs). An ANOVA test was performed to identify statistically significantly changed proteins or genes. (B) Temporal expression levels of selected prognosis signature proteins across 15 time points during liver development. Proteins in blue boxes are correlated with better prognosis; proteins in red boxes are correlated with poor prognosis. iCluster 1–3 subtypes were obtained from the TCGA paper (The Cancer Genome Atlas Research Network 2017); S-I, -II, -III subtypes were obtained from the HCC paper (Jiang et al. 2019). (C) Heatmaps showing temporal expression patterns of S-I-, S-II-, or S-III-enriched proteins across 15 time points during liver development.