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Schematic illustrating the working principle of curcumol related signaling in mouse melanoma B16 cells. We propose that curcumol maintained the level of miR-152-p in mouse melanoma B16 cells, thereby inactivating c-MET/PI3K/AKT-dependent signaling pathway associated with mouse melanoma B16 cell death. In addition, our results also imply that ERK/NF-κB signaling pathway contributes partially in curcumol-mediated effects.
