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. 2020 Feb 7;45(4):1176–1186. doi: 10.3892/ijmm.2020.4493

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Copyright: © Fu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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CORM-3 ameliorates upregulation in pyroptosis-associated protein expression in the hippocampus. (A) Representative western blots of cleaved caspase-1, GSDMD, NLRP3, IL-1β and IL-18 in the hippocampal tissue. (B-F) Relative expression levels of cleaved caspase-1, GSDMD, NLRP3, IL-1β and IL-18 in the hippocampal tissue evaluated by western blotting. Data are presented as the mean ± SD (n=6 per group). ^*P<0.05 vs. Sham; ^$P<0.05 vs. HSR vehicle and HSR iCORM-3. CORM, carbon monoxide-releasing molecule; HSR, hemorrhage shock and resuscitation; iCORM-3, inactive CORM-3; NLRP3, nucleotide-binding oligomerization domain-like receptors pyrin domain-3; IL, interleukin; GSDMD, Gasdermin D.