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. 2020 Mar 3;11(3):167. doi: 10.1038/s41419-020-2356-9

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a–c EdU, clonogenic and MTT assays were performed to measure the effect of HMGA1P6 on ovarian cancer cell proliferation. d Representative tumors in xenografts of A2780 cells with HMGA1P6 overexpression compared to control cells (left panel), immunohistochemical staining of Ki-67 and HMGA1 were performed in tumor tissues (right panel). Data are presented as means ± S.D. *p < 0.05, **p < 0.01.