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. 2020 Jan 10;122(5):705–714. doi: 10.1038/s41416-019-0703-3

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — HCT-116/miR-107-overexpressing cells and HCT-116/vector cells were injected subcutaneously. Both groups received intraperitoneal injections of L-OHP. a Representative photograph of tumours (scale bar: 1 cm). b, c Tumour weight and volume were measured. d, e Immunohistochemistry (IHC) detection of CAB39, p-AMPK and p-mTOR in tumour tissues. Scale bar: 100 µm. n = 6, *P < 0.05, **P < 0.01, ***P < 0.001. f A schematic diagram for illustrating the miR-107/CAB39/AMPK/mTOR pathway partially involved in the effect of DCA on increasing chemosensitivity.