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. 2020 Feb 19;64:10.29219/fnr.v64.3525. doi: 10.29219/fnr.v64.3525

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© 2020 Yuanwei Wang et al.

This is an Open Access article distributed under the terms of the Creative Commons Attribution 4.0 International License, allowing third parties to copy and redistribute the material in any medium or format and to remix, transform, and build upon the material for any purpose, even commercially, provided the original work is properly cited and states its license.

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Fig. 6 — CAP regulated mRNA expressions of FFAR2, gut hormone peptides, gut hormone peptides receptor, and neuropeptides. (A) FFAR2 gene expressions,(B) PYY gene expressions, (C) GLP-1 gene expressions in the colon; (D) PYY-Y2 gene expressions, (E) GLP-1R gene expressions, (F) POMC gene expressions,(G) CART gene expressions, (H) NPY gene expressions, and (I) AgRP gene expressions in the hypothalamus. Results are represented as mean ± SD (n = 6). WTH, WT mice fed with high-fat diet; WTHC, WT mice fed with high-fat diet and CAP; KOS, KO mice fed with low-fat diet; KOH, KO mice fed with high-fat diet; KOHC, KO mice fed with high-fat diet and CAP. a, WTH groupversus WTS group; b, WTHC groupversus WTH group; c, KOH group versus KOS group; d, KOHC groupversus KOH group( one-way ANOVA followed by Dunnett’s test, p < 0.05).