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. 2020 Feb 18;21(4):1941–1949. doi: 10.3892/mmr.2020.10989

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Copyright: © Liu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Alterations to the proportion of apoptotic HaCaT cells in response to TET2 knockdown or overexpression. Proportion of apoptotic cells was (A) determined by flow cytometry and (B) quantified following TET2 knockdown in HaCaT cells. Proportion of apoptotic cells was (C) determined by flow cytometry and (D) quantified following TET2 overexpression in HaCaT cells. Data are presented as the mean ± SD of three independent experiments performed in triplicate. *P<0.05, **P<0.01 vs. NC group. TET2, ten-eleven translocation-2; NC, negative control; PI, propidium iodide; si, small interfering RNA.