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. 2020 Mar 5;10:4106. doi: 10.1038/s41598-020-60456-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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(a) Density of PubMed IDs (PMIDs) per gene for all prognostic unfavorable genes in various types of cancer from the Human Protein Atlas; the bulk of genes have few articles and the density begins to decrease sharply at 100. Genes with ≤50 PMIDs are defined as functionally enigmatic genes; and genes with number of PMIDs >50 are considered well-studied genes. (b,c) Distribution of autosomal dominant and recessive disease associations among functionally enigmatic genes and well-studied genes. (d,e) Distribution of primate-specific genes and conserved eukaryotic genes among functionally enigmatic genes and well-studied genes.