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. 2020 Mar 5;10:4158. doi: 10.1038/s41598-020-60984-6

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Possible mechanisms underlying the GCL2505 effects on host energy metabolism. Number of B. lactis in the gut and the plasma acetate level increase in response to GCL2505. Elevated acetate level modulates host energy metabolism (e.g., suppressed body fat accumulation, increased insulin sensitivity, and enhanced systemic fatty acid metabolism) in a GPR43-dependent manner.