Fig. 5.

Schematic demonstration of efficacy of relaxin to cisplatin-induced testicular dysfunction. RLN administration efficiently scavenged ROS via activation of SOD, CAT, and GPx and upregulation of GSH to prevent lipid peroxidation and decreased apoptosis by altering Bcl2 and Casp3 expression, thereby reducing histopathological damage and restoring spermatogenesis