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. 2020 Mar 10;6:10. doi: 10.1038/s41522-020-0120-7

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a Representative images of alveolar bone loss on the palatal surfaces of maxillary molars in control and infection groups. Scale bar represents 0.2 mm. b The graphs represent alveolar bone loss in control mice or mice with the polymicrobial infection (Infection). Data represent the means ± s.d. from six mice per group. For each mouse, alveolar bone loss was calculated as the average from 28 sites (3 sites on the first molar, 2 sites on the second molar, and 2 sites on the third molar, on both sides of the left maxilla and mandible). *, the difference in alveolar bone loss was significant (p < 0.001) compared to the Control group. c The percentage of intrabony defects was calculated as the number of tooth surfaces containing periodontal intrabony defects out of total tooth surfaces. For each group, there were a total of 72 tooth surfaces (6 mice, 36 molars, 72 sides (buccal, palatal/lingual)). *, the difference in the percentage of intrabony defect was significant (p < 0.001) compared to the Control group.