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. 2020 Mar 10;10:4435. doi: 10.1038/s41598-020-61298-3

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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(A) Heat maps of somatic cell mutation, stage and age information in 10 patients with HCC; (B) left: Highly frequent mutant genes in 10 patients (25 in total). Right: Highly frequent mutant genes in TCGA database (28 in total). Heat maps were generated for the 53 gene mutations in 10 patients. The frequency of TCGA mutations was not high in our 10 patients. (C) Comparison of high frequency gene mutations between 10 HCC patients in our center and TCGA database. (D) GO and KEGG pathways involved in 10 HCC patients in our center. (E) Circos of mutation information in 10 HCC patients. (F) Venn diagram for comparison of mutant genes and TCGA mutant genes in 10 HCC patients. (G) Clustering heat map of high frequency mutant genes in 10 HCC patients. (H) Heat map of driver gene mutation in 10 HCC patients.