Skip to main content
. Author manuscript; available in PMC: 2020 Mar 12.
Published in final edited form as: Int J Obes (Lond). 2019 Feb 18;43(9):1783–1794. doi: 10.1038/s41366-019-0324-1

Figure 2.

Figure 2.

Adipocyte morphology, gene expression, and GAG content with XylT2 deficiency in 2–3 month male mice. Adipocyte morphology was assessed by hematoxylin and eosin staining of GAT. (a) Xylt2+/+ and (b) Xylt2−/− mice followed by (c) adipocyte size distribution measurements, n=4 of each genotype. (d)(e)(f) Similar analyses in MAT, n=4 of each genotype, scale bars = 100μm. (g) GAT expression of adipogenic differentiation factors peroxisomal proliferation activator receptor-γ (Pparg), CCAAT/enhancer binding protein-α (Cebpa), and fatty acid binding protein four (Fabp4) in GAT (n=4 of each genotype). (h) GAT expression analyses of known lipodystrophic genes in Xylt2+/+ and Xylt2−/− mice. * p<0.05.