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. 2020 Feb 19;19(4):3307–3315. doi: 10.3892/ol.2020.11414

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Copyright: © Tian et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — Increased expression levels of ASPM, BUB1B and SPDL1 in PDAC. Red represents tumor tissue and black represents normal tissue. (A) ASPM, (B) BUB1B and (C) SPDL1 were upregulated in the tumors of patients with PDAC and the associations between (D) ASPM, (E) BUB1B and (F) SPDL1 were analyzed via Pearson's correlation analysis. (G) ASPM, (H) BUB1B and (I) SPDL1 were also upregulated in the tumor tissues of patients with advanced PDAC. *P<0.05. ASPM, abnormal spindle-like microcephaly-associated protein; BUB1B, mitotic checkpoint serine/threonine-protein kinase BUB1 β; SPDL1, protein spindly; PDAC, pancreatic ductal adenocarcinoma; TPM, Transcripts Per Kilobase of exon model per Million mapped reads.