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. 2020 Mar 12;16:1744806920911543. doi: 10.1177/1744806920911543

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© The Author(s) 2020

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 5. — Knockdown of GluA1 in central amygdala potentiates morphine withdrawal-induced aversion. CPA behavior in rats with naloxone-precipitated morphine withdrawal (a, n = 8 rats each group) and in rats conditioned with saline and naloxone (b, n = 6 rats each group) after bilateral infusion of AAV-GluA1-shRNA or AAV-GluA1-scrambled (scr) shRNA into CeA. **p < 0.01. ****p < 0.0001 (two-way analysis of variance).