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. 2020 Feb 18;12(2):472. doi: 10.3390/cancers12020472

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© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Tumor markers in YAP-induced HCC mice model treated with pAFP–DTA. Serum concentration of AFP and des-gamma carboxyprothrombin (DCP) quantified at 60 days (a,b), 120 days (c,d), and 180 days (e,f) after YAP-expressing plasmid (5SA) delivery with/without the gene therapy of pAFP–DTA by ELISA. The values represent mean ± standard deviation (n = 5 for each group). * p < 0.05; ** p < 0.01; *** p < 0.001; and N.S. One-way ANOVA followed by Bonferroni’s multiple comparison test. 0 M, 2 M, and 4 M, at 0, 2, and 4 months after the delivery of pAFP–DTA.

Tumor markers in YAP-induced HCC mice model treated with pAFP–DTA. Serum concentration of AFP and des-gamma carboxyprothrombin (DCP) quantified at 60 days (a,b), 120 days (c,d), and 180 days (e,f) after YAP-expressing plasmid (5SA) delivery with/without the gene therapy of pAFP–DTA by ELISA. The values represent mean ± standard deviation (n = 5 for each group). * p < 0.05; ** p < 0.01; *** p < 0.001; and N.S. One-way ANOVA followed by Bonferroni’s multiple comparison test. 0 M, 2 M, and 4 M, at 0, 2, and 4 months after the delivery of pAFP–DTA.